{"id":2178,"date":"2026-07-07T14:51:22","date_gmt":"2026-07-07T12:51:22","guid":{"rendered":"https:\/\/www.amyloidosisalliance.org\/arten-der-amyloidose\/"},"modified":"2026-10-21T17:37:45","modified_gmt":"2026-10-21T15:37:45","slug":"arten-der-amyloidose","status":"publish","type":"page","link":"https:\/\/www.amyloidosisalliance.org\/de\/arten-der-amyloidose\/","title":{"rendered":"Arten der Amyloidose"},"content":{"rendered":"\n<nav aria-label=\"Breadcrumb\" class=\"wp-block-group aa-breadcrumb is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"aa-inner\"><a href=\"\/\">Startseite<\/a><span class=\"aa-breadcrumb__sep\">\u203a<\/span><span class=\"aa-breadcrumb__label\">Amyloidose<\/span><span class=\"aa-breadcrumb__sep\">\u203a<\/span><span class=\"aa-breadcrumb__current\">Arten der Amyloidose<\/span><\/div>\n<\/nav>\n\n<section class=\"wp-block-group aa-page-hero is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-page-hero__grid is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-overline wp-block-paragraph\">Amyloidose<\/p>\n\n\n\n<h1 class=\"wp-block-heading\">Arten der Amyloidose<\/h1>\n\n\n\n<p class=\"aa-lead wp-block-paragraph\">Amyloidose ist keine einheitliche Krankheit, sondern eine Familie unterschiedlicher Erkrankungen, die jeweils durch die Art des fehlgefalteten Proteins und die betroffenen Organe bestimmt werden. Jede dieser Formen zu verstehen, ist der erste Schritt zu einer genauen Diagnose und einer gezielten Behandlung.<\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-page-hero__photo is-layout-flow wp-block-group-is-layout-flow\">\n<figure class=\"wp-block-image size-full\"><img loading=\"lazy\" decoding=\"async\" width=\"1080\" height=\"1350\" src=\"https:\/\/www.amyloidosisalliance.org\/wp-content\/uploads\/2026\/09\/Design-sans-titre.zip-6.jpg\" alt=\"\" class=\"wp-image-1190\" srcset=\"https:\/\/www.amyloidosisalliance.org\/wp-content\/uploads\/2026\/09\/Design-sans-titre.zip-6.jpg 1080w, https:\/\/www.amyloidosisalliance.org\/wp-content\/uploads\/2026\/09\/Design-sans-titre.zip-6-240x300.jpg 240w, https:\/\/www.amyloidosisalliance.org\/wp-content\/uploads\/2026\/09\/Design-sans-titre.zip-6-819x1024.jpg 819w, https:\/\/www.amyloidosisalliance.org\/wp-content\/uploads\/2026\/09\/Design-sans-titre.zip-6-768x960.jpg 768w\" sizes=\"auto, (max-width: 1080px) 100vw, 1080px\" \/><\/figure>\n<\/div>\n<\/div>\n<\/div>\n<\/section>\n\n<nav aria-label=\"Section navigation\" class=\"wp-block-group aa-anchorband is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-columns aa-anchor-cols is-layout-flex wp-container-core-columns-is-layout-b2e659ba wp-block-columns-is-layout-flex\">\n<div class=\"wp-block-column aa-anchor-item is-layout-flow wp-block-column-is-layout-flow\">\n<p class=\"num wp-block-paragraph\">01<\/p>\n\n\n\n<p class=\"lbl wp-block-paragraph\"><a href=\"#diversity\">Die Vielfalt<\/a><\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-column aa-anchor-item is-layout-flow wp-block-column-is-layout-flow\">\n<p class=\"num wp-block-paragraph\">02<\/p>\n\n\n\n<p class=\"lbl wp-block-paragraph\"><a href=\"#al\">AL-Amyloidose<\/a><\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-column aa-anchor-item is-layout-flow wp-block-column-is-layout-flow\">\n<p class=\"num wp-block-paragraph\">03<\/p>\n\n\n\n<p class=\"lbl wp-block-paragraph\"><a href=\"#attr\">ATTR-Amyloidose<\/a><\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-column aa-anchor-item is-layout-flow wp-block-column-is-layout-flow\">\n<p class=\"num wp-block-paragraph\">04<\/p>\n\n\n\n<p class=\"lbl wp-block-paragraph\"><a href=\"#aa\">AA-Amyloidose<\/a><\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-column aa-anchor-item is-layout-flow wp-block-column-is-layout-flow\">\n<p class=\"num wp-block-paragraph\">05<\/p>\n\n\n\n<p class=\"lbl wp-block-paragraph\"><a href=\"#rarer\">Seltenere Formen<\/a><\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-column aa-anchor-item is-layout-flow wp-block-column-is-layout-flow\">\n<p class=\"num wp-block-paragraph\">06<\/p>\n\n\n\n<p class=\"lbl wp-block-paragraph\"><a href=\"#localized\">Lokalisiert<\/a><\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-column aa-anchor-item is-layout-flow wp-block-column-is-layout-flow\">\n<p class=\"num wp-block-paragraph\">07<\/p>\n\n\n\n<p class=\"lbl wp-block-paragraph\"><a href=\"#compare\">Formen vergleichen<\/a><\/p>\n<\/div>\n<\/div>\n<\/div>\n<\/nav>\n\n<section id=\"diversity\" class=\"wp-block-group aa-section aa-section--white aa-intro is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-section-num wp-block-paragraph\">01<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Die gro\u00dfe Vielfalt der Amyloidose<\/h2>\n\n\n\n<p class=\"wp-block-paragraph\">Von mehr als 40 menschlichen Vorl\u00e4uferproteinen ist bekannt, dass sie Amyloidablagerungen bilden. Die Amyloidose wird nach dem Protein eingeteilt, aus dem die Fibrillen bestehen. Die h\u00e4ufigsten systemischen Formen, AL, ATTR und AA, beruhen jeweils auf unterschiedlichen Vorl\u00e4uferproteinen, betreffen unterschiedliche Organsysteme und erfordern grundlegend verschiedene Behandlungsstrategien.<\/p>\n\n\n\n<p class=\"wp-block-paragraph\">Da die Symptome oft denen h\u00e4ufigerer Krankheiten \u00e4hneln, ist eine korrekte Typisierung unerl\u00e4sslich und erfordert spezialisierte Untersuchungen. Eine fr\u00fche und genaue Typisierung ist die Grundlage einer wirksamen Versorgung.<\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-intro__media is-layout-flow wp-block-group-is-layout-flow\">\n<figure class=\"aa-protein\" role=\"img\" aria-label=\"Illustration: Ein korrekt gefaltetes Protein faltet sich fehlerhaft und lagert sich zu Amyloidfibrillen zusammen\">\n\t\t\t\t<svg viewbox=\"0 0 480 360\" aria-hidden=\"true\" focusable=\"false\">\n\t\t\t\t\t<defs>\n\t\t\t\t\t\t<lineargradient id=\"aa-prot-core\" x1=\"0\" y1=\"0\" x2=\"1\" y2=\"1\">\n\t\t\t\t\t\t\t<stop offset=\"0%\" stop-color=\"#167a82\"><\/stop>\n\t\t\t\t\t\t\t<stop offset=\"100%\" stop-color=\"#00666d\"><\/stop>\n\t\t\t\t\t\t<\/lineargradient>\n\t\t\t\t\t\t<lineargradient id=\"aa-prot-fib\" x1=\"0\" y1=\"0\" x2=\"1\" y2=\"0\">\n\t\t\t\t\t\t\t<stop offset=\"0%\" stop-color=\"#f6a955\"><\/stop>\n\t\t\t\t\t\t\t<stop offset=\"100%\" stop-color=\"#f39325\"><\/stop>\n\t\t\t\t\t\t<\/lineargradient>\n\t\t\t\t\t<\/defs>\n\n\t\t\t\t\t<!-- Prot\u00e9ine correctement repli\u00e9e : h\u00e9lice compacte -->\n\t\t\t\t\t<g transform=\"translate(96 130)\">\n\t\t\t\t\t\t<circle r=\"62\" fill=\"#eaf3f3\"><\/circle>\n\t\t\t\t\t\t<path d=\"M-34 26 C-46 2 -26 -14 -6 -8 C14 -2 30 -16 22 -34\" fill=\"none\" stroke=\"url(#aa-prot-core)\" stroke-width=\"9\" stroke-linecap=\"round\"><\/path>\n\t\t\t\t\t\t<path d=\"M-26 40 C-4 44 18 30 26 10\" fill=\"none\" stroke=\"url(#aa-prot-core)\" stroke-width=\"9\" stroke-linecap=\"round\" opacity=\"0.55\"><\/path>\n\t\t\t\t\t\t<circle cx=\"22\" cy=\"-34\" r=\"7\" fill=\"#00666d\"><\/circle>\n\t\t\t\t\t\t<circle cx=\"-34\" cy=\"26\" r=\"7\" fill=\"#00666d\"><\/circle>\n\t\t\t\t\t<\/g>\n\n\t\t\t\t\t<!-- Transition -->\n\t\t\t\t\t<g stroke=\"#c9d2d6\" stroke-width=\"2\" stroke-linecap=\"round\">\n\t\t\t\t\t\t<line x1=\"176\" y1=\"130\" x2=\"212\" y2=\"130\" stroke-dasharray=\"2 8\"><\/line>\n\t\t\t\t\t\t<path d=\"M206 122 L216 130 L206 138\" fill=\"none\"><\/path>\n\t\t\t\t\t<\/g>\n\n\t\t\t\t\t<!-- Prot\u00e9ine mal repli\u00e9e : feuillets b\u00eata empil\u00e9s -->\n\t\t\t\t\t<g transform=\"translate(300 130)\">\n\t\t\t\t\t\t<g fill=\"none\" stroke=\"url(#aa-prot-fib)\" stroke-width=\"8\" stroke-linecap=\"round\">\n\t\t\t\t\t\t\t<path d=\"M-56 -34 L-20 -34 L-20 -14 L16 -14 L16 -34 L52 -34\"><\/path>\n\t\t\t\t\t\t\t<path d=\"M-56 -6 L-20 -6 L-20 14 L16 14 L16 -6 L52 -6\"><\/path>\n\t\t\t\t\t\t\t<path d=\"M-56 22 L-20 22 L-20 42 L16 42 L16 22 L52 22\"><\/path>\n\t\t\t\t\t\t<\/g>\n\t\t\t\t\t<\/g>\n\n\t\t\t\t\t<!-- Fibrille amylo\u00efde : les feuillets s'assemblent en filament -->\n\t\t\t\t\t<g transform=\"translate(240 268)\" fill=\"none\" stroke-linecap=\"round\">\n\t\t\t\t\t\t<path d=\"M-190 0 C-150 -22 -110 22 -70 0 C-30 -22 10 22 50 0 C90 -22 130 22 170 0\" stroke=\"url(#aa-prot-fib)\" stroke-width=\"7\"><\/path>\n\t\t\t\t\t\t<path d=\"M-190 14 C-150 -8 -110 36 -70 14 C-30 -8 10 36 50 14 C90 -8 130 36 170 14\" stroke=\"url(#aa-prot-fib)\" stroke-width=\"7\" opacity=\"0.45\"><\/path>\n\t\t\t\t\t<\/g>\n\n\t\t\t\t\t<text x=\"96\" y=\"222\" text-anchor=\"middle\" fill=\"#5a6a73\" font-family=\"'JetBrains Mono', ui-monospace, monospace\" font-size=\"12\" letter-spacing=\"1.4\">GEFALTET<\/text>\n\t\t\t\t\t<text x=\"300\" y=\"222\" text-anchor=\"middle\" fill=\"#5a6a73\" font-family=\"'JetBrains Mono', ui-monospace, monospace\" font-size=\"12\" letter-spacing=\"1.4\">FEHLGEFALTET<\/text>\n\t\t\t\t\t<text x=\"240\" y=\"320\" text-anchor=\"middle\" fill=\"#5a6a73\" font-family=\"'JetBrains Mono', ui-monospace, monospace\" font-size=\"12\" letter-spacing=\"1.4\">AMYLOIDFIBRILLE<\/text>\n\t\t\t\t<\/svg>\n\t\t\t<\/figure>\n<\/div>\n<\/div>\n<\/section>\n\n<section id=\"al\" class=\"wp-block-group aa-section aa-section--cream aa-type is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-type-split aa-type-split--photo-left is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-type-photo is-layout-flow wp-block-group-is-layout-flow\">\n<figure class=\"wp-block-image size-full\"><img decoding=\"async\" src=\"\/wp-content\/themes\/amyloidosis-alliance\/assets\/images\/disease\/al-card.svg\" alt=\"\"\/><\/figure>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-type-text is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-type-label wp-block-paragraph\">02 \u00b7 Systemisch<\/p>\n\n\n\n<h2 class=\"wp-block-heading aa-type-h\">AL-Amyloidose<\/h2>\n\n\n\n<p class=\"aa-type-p wp-block-paragraph\">Die AL-Amyloidose (Leichtketten-Amyloidose) entsteht durch eine krankhafte Vermehrung von Plasmazellen im Knochenmark, die fehlgefaltete Immunglobulin-Leichtketten bilden. Diese Fragmente lagern sich als Amyloidfibrillen vor allem im Herzen und in den Nieren ab, k\u00f6nnen aber jedes Organ befallen.<\/p>\n\n\n\n<p class=\"aa-type-p wp-block-paragraph\">Sie ist die h\u00e4ufigste Form der systemischen Amyloidose in den Industriel\u00e4ndern. Die Behandlung richtet sich gegen die zugrunde liegende Plasmazellerkrankung, in der Regel mit Chemotherapieprotokollen, die denen beim multiplen Myelom \u00e4hneln.<\/p>\n\n\n\n<div class=\"wp-block-buttons is-layout-flex wp-block-buttons-is-layout-flex\">\n<div class=\"wp-block-button aa-btn aa-btn--teal\"><a class=\"wp-block-button__link wp-element-button\" href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/al-amyloidosis\/\">AL-Amyloidose entdecken \u2192<\/a><\/div>\n<\/div>\n<\/div>\n<\/div>\n<\/div>\n<\/section>\n\n<section id=\"attr\" class=\"wp-block-group aa-section aa-section--white aa-type is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-type-split aa-type-split--photo-right is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-section-num wp-block-paragraph\">03 \u00b7 Systemisch<\/p>\n\n\n\n<h2 class=\"wp-block-heading aa-type-h\">ATTR-Amyloidose<\/h2>\n\n\n\n<p class=\"aa-type-p wp-block-paragraph\">Die ATTR-Amyloidose (Transthyretin-Amyloidose) entsteht, wenn das in der Leber gebildete Protein Transthyretin instabil wird und sich fehlerhaft faltet. Sie betrifft vor allem das Herz und das periphere Nervensystem und tritt in zwei verschiedenen Formen auf, mit unterschiedlichen Ursachen und Behandlungswegen.<\/p>\n\n\n\n<div class=\"wp-block-group aa-subtype-list is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-subtype-row is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-subtype-row__name wp-block-paragraph\">Wild-type ATTR<\/p>\n\n\n\n<p class=\"aa-subtype-row__desc wp-block-paragraph\">Tritt spontan mit dem Alter auf und ist nicht erblich. Zeigt sich meist als Kardiomyopathie bei \u00e4lteren M\u00e4nnern. Wird dank besserer kardialer Bildgebung immer h\u00e4ufiger erkannt.<\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-buttons is-layout-flex wp-block-buttons-is-layout-flex\">\n<div class=\"wp-block-button aa-btn aa-btn--teal aa-btn--sm\"><a class=\"wp-block-button__link wp-element-button\" href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/attrwt-amyloidosis\/\">Entdecken \u2192<\/a><\/div>\n<\/div>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-subtype-row is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-subtype-row__name wp-block-paragraph\">Heredit\u00e4re ATTR<\/p>\n\n\n\n<p class=\"aa-subtype-row__desc wp-block-paragraph\">Verursacht durch eine genetische Mutation im TTR-Gen. Die Vererbung erfolgt autosomal-dominant. Erkrankungsbeginn und Organbeteiligung h\u00e4ngen von der jeweiligen Mutation ab; die Neuropathie ist ein typisches Merkmal.<\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-buttons is-layout-flex wp-block-buttons-is-layout-flex\">\n<div class=\"wp-block-button aa-btn aa-btn--teal aa-btn--sm\"><a class=\"wp-block-button__link wp-element-button\" href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/attrv-amyloidosis\/\">Entdecken \u2192<\/a><\/div>\n<\/div>\n<\/div>\n<\/div>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-type-photo is-layout-flow wp-block-group-is-layout-flow\">\n<figure class=\"wp-block-image size-full\"><img decoding=\"async\" src=\"\/wp-content\/themes\/amyloidosis-alliance\/assets\/images\/disease\/attr-card.svg\" alt=\"\"\/><\/figure>\n<\/div>\n<\/div>\n<\/div>\n<\/section>\n\n<section id=\"aa\" class=\"wp-block-group aa-section aa-section--cream aa-type is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-type-split aa-type-split--photo-left is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-type-photo is-layout-flow wp-block-group-is-layout-flow\">\n<figure class=\"wp-block-image size-full\"><img decoding=\"async\" src=\"\/wp-content\/themes\/amyloidosis-alliance\/assets\/images\/disease\/aa-card.svg\" alt=\"\"\/><\/figure>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-type-text is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-type-label wp-block-paragraph\">04 \u00b7 Systemisch<\/p>\n\n\n\n<h2 class=\"wp-block-heading aa-type-h\">AA-Amyloidose<\/h2>\n\n\n\n<p class=\"aa-type-p wp-block-paragraph\">Die AA-Amyloidose entsteht als Folgekomplikation einer anhaltenden systemischen Entz\u00fcndung. Das verantwortliche Protein, Serum-Amyloid A (SAA), ist ein Akute-Phase-Protein, das die Leber bei chronisch-entz\u00fcndlichen Zust\u00e4nden bildet, etwa bei rheumatoider Arthritis, chronisch-entz\u00fcndlichen Darmerkrankungen oder chronischen Infektionen.<\/p>\n\n\n\n<p class=\"aa-type-p wp-block-paragraph\">Haupts\u00e4chlich betroffen sind die Nieren, mit Proteinurie und fortschreitendem Nierenversagen. Die Behandlung zielt darauf ab, die zugrunde liegende entz\u00fcndliche oder infekti\u00f6se Erkrankung zu kontrollieren und die Bildung von Serum-Amyloid A (SAA) zu unterdr\u00fccken.<\/p>\n\n\n\n<div class=\"wp-block-buttons is-layout-flex wp-block-buttons-is-layout-flex\">\n<div class=\"wp-block-button aa-btn aa-btn--teal\"><a class=\"wp-block-button__link wp-element-button\" href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/aa-amyloidosis\/\">AA-Amyloidose entdecken \u2192<\/a><\/div>\n<\/div>\n<\/div>\n<\/div>\n<\/div>\n<\/section>\n\n<section id=\"rarer\" class=\"wp-block-group aa-section aa-section--white aa-type is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-type-split aa-type-split--photo-right is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-section-num wp-block-paragraph\">05<\/p>\n\n\n\n<h2 class=\"wp-block-heading aa-type-h\">Seltenere erbliche Formen<\/h2>\n\n\n\n<p class=\"aa-type-p wp-block-paragraph\">Neben der ATTR-Amyloidose verursachen weitere Genmutationen seltenere erbliche Formen der systemischen Amyloidose. Jede wird durch ihr Vorl\u00e4uferprotein bestimmt und hat ein eigenes Muster der Organbeteiligung. Ein Gentest ist f\u00fcr die Diagnose unerl\u00e4sslich und hat Folgen f\u00fcr die Familienangeh\u00f6rigen.<\/p>\n\n\n\n<div class=\"wp-block-group aa-subtype-list is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-rare-row is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-rare-row__name wp-block-paragraph\">AAPOA1<\/p>\n\n\n\n<p class=\"aa-rare-row__desc wp-block-paragraph\">Mutation des Apolipoproteins A-I. Betrifft Nieren, Leber und periphere Nerven. Autosomal-dominante Vererbung.<\/p>\n\n\n\n<div class=\"wp-block-buttons is-layout-flex wp-block-buttons-is-layout-flex\">\n<div class=\"wp-block-button aa-btn aa-btn--teal aa-btn--sm\"><a class=\"wp-block-button__link wp-element-button\" href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/rare-amyloidosis\/\">Entdecken \u2192<\/a><\/div>\n<\/div>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-rare-row is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-rare-row__name wp-block-paragraph\">AGEL<\/p>\n\n\n\n<p class=\"aa-rare-row__desc wp-block-paragraph\">Mutation von Gelsolin. Verursacht vor allem eine Hirnnervenneuropathie und eine gittrige Hornhautdystrophie. Am h\u00e4ufigsten in Finnland.<\/p>\n\n\n\n<div class=\"wp-block-buttons is-layout-flex wp-block-buttons-is-layout-flex\">\n<div class=\"wp-block-button aa-btn aa-btn--teal aa-btn--sm\"><a class=\"wp-block-button__link wp-element-button\" href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/rare-amyloidosis\/\">Entdecken \u2192<\/a><\/div>\n<\/div>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-rare-row is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-rare-row__name wp-block-paragraph\">ALYS<\/p>\n\n\n\n<p class=\"aa-rare-row__desc wp-block-paragraph\">Mutation des Lysozyms. Vor allem Nierenbeteiligung, mit Ablagerungen in der Leber. Historisch wurden F\u00e4lle in Gro\u00dfbritannien beschrieben.<\/p>\n\n\n\n<div class=\"wp-block-buttons is-layout-flex wp-block-buttons-is-layout-flex\">\n<div class=\"wp-block-button aa-btn aa-btn--teal aa-btn--sm\"><a class=\"wp-block-button__link wp-element-button\" href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/rare-amyloidosis\/\">Entdecken \u2192<\/a><\/div>\n<\/div>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-rare-row is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-rare-row__name wp-block-paragraph\">AFib<\/p>\n\n\n\n<p class=\"aa-rare-row__desc wp-block-paragraph\">Mutation der Fibrinogen-A-alpha-Kette. Ausschlie\u00dflich Nierenerkrankung; eine der h\u00e4ufigeren erblichen Formen in Westeuropa.<\/p>\n\n\n\n<div class=\"wp-block-buttons is-layout-flex wp-block-buttons-is-layout-flex\">\n<div class=\"wp-block-button aa-btn aa-btn--teal aa-btn--sm\"><a class=\"wp-block-button__link wp-element-button\" href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/rare-amyloidosis\/\">Entdecken \u2192<\/a><\/div>\n<\/div>\n<\/div>\n<\/div>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-type-photo is-layout-flow wp-block-group-is-layout-flow\">\n<figure class=\"wp-block-image size-full\"><img decoding=\"async\" src=\"\/wp-content\/themes\/amyloidosis-alliance\/assets\/images\/disease\/rare-card.svg\" alt=\"\"\/><\/figure>\n<\/div>\n<\/div>\n<\/div>\n<\/section>\n\n<section id=\"localized\" class=\"wp-block-group aa-section aa-section--forest aa-type is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-type-split aa-type-split--photo-left is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-type-photo is-layout-flow wp-block-group-is-layout-flow\">\n<figure class=\"wp-block-image size-full\"><img decoding=\"async\" src=\"\/wp-content\/themes\/amyloidosis-alliance\/assets\/images\/disease\/localised-card.svg\" alt=\"\"\/><\/figure>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-type-text is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-type-label wp-block-paragraph\">06 \u00b7 Lokalisiert<\/p>\n\n\n\n<h2 class=\"wp-block-heading aa-type-h\">Lokalisierte Amyloidose<\/h2>\n\n\n\n<p class=\"aa-type-p wp-block-paragraph\">Anders als die systemischen Formen ist die lokalisierte Amyloidose auf ein einzelnes Organ oder Gewebe beschr\u00e4nkt, am h\u00e4ufigsten die Atemwege, die Haut, die Harnblase oder die Augenh\u00f6hle. Das Amyloid wird vor Ort von ans\u00e4ssigen Plasmazellen gebildet und breitet sich nicht im K\u00f6rper aus.<\/p>\n\n\n\n<p class=\"aa-type-p wp-block-paragraph\">Die Prognose ist im Allgemeinen g\u00fcnstiger als bei systemischen Formen, da die Funktion lebenswichtiger Organe nur selten beeintr\u00e4chtigt ist. Die Behandlung richtet sich nach der betroffenen Stelle und dem Ausma\u00df der lokalen Gewebesch\u00e4digung.<\/p>\n\n\n\n<div class=\"wp-block-buttons is-layout-flex wp-block-buttons-is-layout-flex\">\n<div class=\"wp-block-button aa-btn aa-btn--primary\"><a class=\"wp-block-button__link wp-element-button\" href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/localised-amyloidosis\/\">Entdecken \u2192<\/a><\/div>\n<\/div>\n<\/div>\n<\/div>\n<\/div>\n<\/section>\n\n<section id=\"compare\" class=\"wp-block-group aa-section aa-section--cream aa-compare-sec is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-centered-head is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-section-num wp-block-paragraph\">07<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Formen vergleichen<\/h2>\n\n\n\n<div class=\"aa-divider-dot\"><\/div>\n\n\n\n<p class=\"wp-block-paragraph\">Ein schneller \u00dcberblick \u00fcber die wichtigsten Arten der Amyloidose: Protein, haupts\u00e4chlich betroffene Organe, Behandlungsansatz und wo Sie mehr erfahren.<\/p>\n<\/div>\n\n\n\n<figure class=\"wp-block-table aa-compare\"><table><thead><tr><th>Form<\/th><th>Protein<\/th><th>Haupts\u00e4chlich betroffene Organe<\/th><th>Behandlungsansatz<\/th><th>Mehr erfahren<\/th><\/tr><\/thead><tbody><tr><td>AL<\/td><td>Immunglobulin-Leichtketten<\/td><td>Herz, Nieren, Leber, Nerven, Weichteile<\/td><td>Gegen die Plasmazellen gerichtete Chemotherapie; Stammzelltransplantation bei geeigneten Patienten<\/td><td><a class=\"aa-compare__link\" href=\"https:\/\/www.amyloidosisalliance.org\/de\/arten-der-amyloidose\/al-amyloidose\/\">AL-Amyloidose \u2192<\/a><\/td><\/tr><tr><td>ATTR, wild-type<\/td><td>Transthyretin (wild-type)<\/td><td>Herz (Kardiomyopathie)<\/td><td>Stabilisierung des TTR und unterst\u00fctzende kardiologische Behandlung; zugelassene Indikationen und Verf\u00fcgbarkeit unterscheiden sich von Land zu Land.<\/td><td><a class=\"aa-compare__link\" href=\"https:\/\/www.amyloidosisalliance.org\/de\/arten-der-amyloidose\/attrwt-amyloidose\/\">ATTRwt-Amyloidose \u2192<\/a><\/td><\/tr><tr><td>ATTR, heredit\u00e4r<\/td><td>Transthyretin (mutiert)<\/td><td>Periphere Nerven, Herz (je nach Mutation)<\/td><td>TTR-Silencer (siRNA, ASO); Stabilisatoren; Lebertransplantation (bei bestimmten Mutationen)<\/td><td><a class=\"aa-compare__link\" href=\"https:\/\/www.amyloidosisalliance.org\/de\/arten-der-amyloidose\/attrv-amyloidose\/\">ATTRv-Amyloidose \u2192<\/a><\/td><\/tr><tr><td>AA<\/td><td>Serum-Amyloid A (SAA)<\/td><td>Nieren, Leber, Milz<\/td><td>Behandlung der zugrunde liegenden Entz\u00fcndungskrankheit; IL-1-Hemmer<\/td><td><a class=\"aa-compare__link\" href=\"https:\/\/www.amyloidosisalliance.org\/de\/arten-der-amyloidose\/aa-amyloidose\/\">AA-Amyloidose \u2192<\/a><\/td><\/tr><tr><td>Seltenere erbliche Formen<\/td><td>ApoA-I, Gelsolin, Lysozym, Fibrinogen\u2026<\/td><td>Je nach Form (Nieren, Nerven, Leber)<\/td><td>Unterst\u00fctzende Behandlung; Organtransplantation in ausgew\u00e4hlten F\u00e4llen<\/td><td><a class=\"aa-compare__link\" href=\"https:\/\/www.amyloidosisalliance.org\/de\/arten-der-amyloidose\/seltene-erbliche-amyloidosen\/\">Seltene erbliche Amyloidosen \u2192<\/a><\/td><\/tr><\/tbody><\/table><\/figure>\n<\/div>\n<\/section>\n\n<section class=\"wp-block-group aa-next aa-next--split is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-inner is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-next__head is-layout-flow wp-block-group-is-layout-flow\">\n<p class=\"aa-section-num wp-block-paragraph\">Weiterlesen<\/p>\n\n\n\n<h2 class=\"wp-block-heading\">Weiterlesen<\/h2>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-next__grid is-layout-flow wp-block-group-is-layout-flow\">\n<div class=\"wp-block-group aa-next-card is-layout-flow wp-block-group-is-layout-flow\">\n<h3 class=\"wp-block-heading\"><a href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/al-amyloidosis\/\">AL-Amyloidose<\/a><\/h3>\n\n\n\n<p class=\"wp-block-paragraph\">Die h\u00e4ufigste systemische Form in westlichen L\u00e4ndern, verursacht durch fehlgefaltete Immunglobulin-Leichtketten.<\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-next-card is-layout-flow wp-block-group-is-layout-flow\">\n<h3 class=\"wp-block-heading\"><a href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/attr-amyloidosis\/\">ATTR-Amyloidose<\/a><\/h3>\n\n\n\n<p class=\"wp-block-paragraph\">Die wild-type und die erbliche Form der Transthyretin-Amyloidose und was sie voneinander unterscheidet.<\/p>\n<\/div>\n\n\n\n<div class=\"wp-block-group aa-next-card is-layout-flow wp-block-group-is-layout-flow\">\n<h3 class=\"wp-block-heading\"><a href=\"https:\/\/www.amyloidosisalliance.org\/types-of-amyloidosis\/rare-amyloidosis\/\">Seltene erbliche Amyloidosen<\/a><\/h3>\n\n\n\n<p class=\"wp-block-paragraph\">Die selteneren erblichen Formen, die beteiligten Proteine und die Organe, die sie typischerweise betreffen.<\/p>\n<\/div>\n<\/div>\n<\/div>\n<\/section>\n","protected":false},"excerpt":{"rendered":"<p>Startseite\u203aAmyloidose\u203aArten der Amyloidose Amyloidose Arten der Amyloidose Amyloidose ist keine einheitliche Krankheit, sondern eine Familie unterschiedlicher Erkrankungen, die jeweils durch die Art des fehlgefalteten Proteins und die betroffenen Organe bestimmt werden. Jede dieser Formen zu verstehen, ist der erste Schritt zu einer genauen Diagnose und einer gezielten Behandlung. 01 Die Vielfalt 02 AL-Amyloidose 03 ATTR-Amyloidose [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"parent":0,"menu_order":0,"comment_status":"closed","ping_status":"closed","template":"","meta":{"footnotes":""},"class_list":["post-2178","page","type-page","status-publish","hentry"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v28.6 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Arten der Amyloidose: AL, AA, ATTR und seltene Formen<\/title>\n<meta name=\"description\" content=\"Amyloidose ist nicht eine Krankheit, sondern eine Familie von Krankheiten. 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